Scientific Programme

Professor
Pharmacology & Pharmaceutical Sciences, School of Pharmacy
University of Southern California, 1985 Zonal Avenue
Los Angeles, CA 90089-9121
Tel: 323 442 1418
Fax: 323 224 7473
E-mail: cadenas@usc.edu

Enrique Cadenas, M.D., Ph.D., graduated in Medicine at the University of Buenos Aires, Argentina and obtained his PhD in Biochemistry at the same institution. He did post-doctoral work at the Johnson Research Foundation, University of Pennsylvania and at the Department of Physiological Chemistry, University of Düsseldorf, Germany. He was associate professor of Pathology at the University of Linköping, Sweden. Currently, Enrique Cadenas is the Charles Krown professor of Pharmacology & Pharmaceutical Sciences at the University of Southern California School of Pharmacy, CA, USA. His laboratory investigates the molecular mechanisms inherent in brain aging and neurodegenerative diseases, with emphasis on Alzheimer’s disease. The research programs attempt to link mitochondrion-centric processes in aging and neurodegeneration to inflammatory responses through redox biology, mainly oxidant-driven cross-talk between metabolism and inflammation.

Professor
Pharmacology & Pharmaceutical Sciences, School of Pharmacy
University of Southern California, 1985 Zonal Avenue
Los Angeles, CA 90089-9121
Tel: 323 442 1418
Fax: 323 224 7473
E-mail: cadenas@usc.edu

Enrique Cadenas, M.D., Ph.D., graduated in Medicine at the University of Buenos Aires, Argentina and obtained his PhD in Biochemistry at the same institution. He did post-doctoral work at the Johnson Research Foundation, University of Pennsylvania and at the Department of Physiological Chemistry, University of Düsseldorf, Germany. He was associate professor of Pathology at the University of Linköping, Sweden. Currently, Enrique Cadenas is the Charles Krown professor of Pharmacology & Pharmaceutical Sciences at the University of Southern California School of Pharmacy, CA, USA. His laboratory investigates the molecular mechanisms inherent in brain aging and neurodegenerative diseases, with emphasis on Alzheimer’s disease. The research programs attempt to link mitochondrion-centric processes in aging and neurodegeneration to inflammatory responses through redox biology, mainly oxidant-driven cross-talk between metabolism and inflammation.